Physical activity and cancer: movement matters.
(2015)
Presentation / Conference Contribution
Campbell, A. (2015, December). Physical activity and cancer: movement matters. Presented at Living with and beyond cancer: a masterclass for primary care
Exercise and cancer survivorship: from evidence to practice. (2015)
Presentation / Conference Contribution
Campbell, A., & Courneya, K. (2015, December). Exercise and cancer survivorship: from evidence to practice. Paper presented at BASES conference 2015
The influence of age and cardiorespiratory fitness on circulating angiogenic T cells and CXCR4 expression. (2015)
Presentation / Conference Contribution
Ross, M. D., Malone, E. M., Simpson, R. J., & Florida-James, G. (2015, May). The influence of age and cardiorespiratory fitness on circulating angiogenic T cells and CXCR4 expression. Presented at Annual Congress of the American College of Sports Medicine (ACSM)Aging is associated with endothelial dysfunction, reduced angiogenic capabilities, and an increase in arterial stiffness. Additionally, regular exercise is known to offset the age-related decline in blood vessel number and function. Circulating angio... Read More about The influence of age and cardiorespiratory fitness on circulating angiogenic T cells and CXCR4 expression..
Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1: SOCS2 REGULATION OF GH INDUCED GROWTH (2015)
Journal Article
Dobie, R., Ahmed, S. F., Staines, K. A., Pass, C., Jasim, S., MacRae, V. E., & Farquharson, C. (2015). Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1: SOCS2 REGULATION OF GH INDUCED GROWTH. Journal of Cellular Physiology, 230(11), 2796-2806. https://doi.org/10.1002/jcp.25006Growth hormone (GH) signaling is essential for postnatal linear bone growth, but the relative importance of GHs actions on the liver and/or growth plate cartilage remains unclear. The importance of liver derived insulin like-growth factor-1 (IGF-1) f... Read More about Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1: SOCS2 REGULATION OF GH INDUCED GROWTH.